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Higher plant densities promoted shoot N uptake and reduced soil mineral N residues and apparent N surplus in root zone.
In both complexes the loop3 regions (with N residues corresponding to N51 in hAID) make either direct or water-mediated hydrogen bonds with the phosphate backbone of ssDNA substrate.
Thus, given a peptide of n residues, the peptide modeling was defined as a success if the accuracy of the ensemble is less than ({mathrm{rmsd}}_{mathrm{C}}({mathrm{n}})).
This is similar to the findings of Gentile et al. [5], who demonstrated that fertiliser N retention was higher in soil amended with low quality (i.e. low N) residue than high N residues, due to net N immobilisation.
The increasing application rate of N fertilizer had no discernible increase on yield parameters, which however resulted in substantial N volatilization and N residues in soils [15, 18, 29].
Indeed, Gentile et al. [5] found that incorporating low quality (i.e. low % N) maize residues could immobilise fertiliser N in field soil and reduce N leaching losses to a greater extent than high quality (high % N) residues, without impacting on crop N uptake over the growing season.
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Expression vectors (pME18) for Flag epitope-tagged forms of human MST1, MST1-N (residues 1 326), MST1-C (residues 327 487), HA-RASSF1A and 3X-IRS reporters have been described previously [26], [27], [28], [29].
However, γ-tubulin was not co-precipitated by either the N- (residues 572 634) or C- (residues 635 660) terminal fragment, suggesting that neither region alone was sufficient for binding.
More specifically, as seen in Figure 5, based on the Cα and Hα chemical shift deviations, it appears that in V22-SH3, the N- (residues 1 6) and C- (residue 47 57) termini are largely unstructured, without any significant secondary structure populated.
More specifically, in V22-SH3, the N- (residues 1 6) and C- (residue 47 57) termini are largely unstructured, without any significant secondary structure populated.
We previously used the N-terminal domain of PABPN1 (PABPN1-N: residues 1-125 of 306) to monitor fibril formation in vitro.
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