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The major cause of left ventricular hypertrophy and failure and the most common problem directly affecting myocardial function is fluid overload and, usually, hypertension.
For patients with acute coronary syndrome (ACS), the key to increased survival and the preservation of myocardial function is rapid diagnosis followed by appropriate early intervention, which will enable timely management strategies and intervention to be carried out.
Intrinsic depression of RV myocardial function is detected as RV hypokinesia, and semi-quantitatively appreciated as a variable degree of RV dilatation (with RV end-diastolic area, RVEDA, to LV end-diastolic area, LVEDA, ratio measurement in a four chamber view).
Support for this mechanism comes from our studies demonstrating that myocardial function is enhanced and myocardial infarct size is significantly decreased and that these alterations are evident and stable for at least 28 days post-injection, the duration of our current studies [19, 34].
The secondary substrate available to generate ATP for myocardial function is glucose.
Myocardial function is depressed even in the early hyperdynamic phase of sepsis [ 53].
Similar(42)
Myocardial function was evaluated using echocardiography.
Myocardial function was evaluated using echocardiography and edge-detection.
Myocardial function was assessed using echocardiography and an Ion-Optix MyoCam system.
Cardiomyocyte contractile and Ca2+ transient properties were evaluated and myocardial function was assessed using echocardiography.
Serum and myocardial carnitine levels in the animals with diabetes and reduced myocardial function were low.
More suggestions(15)
myocardial thickness is
myocardial tagging is
myocardial differentiation is
myocardial function deteriorates
myocardial migration is
myocardial necrosis is
myocardial infarction is
myocardial dysfunction is
myocardial fibrosis is
myocardial hypertrophy is
myocardial injury is
myocardial depression is
myocardial involvement is
myocardial ischaemia is
myocardial aggression is
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