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Myocardial function deteriorates during ventricular remodeling in patients with congestive heart failure (HF).
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Assessment of myocardial longitudinal function has proved to be a sensitive marker of deteriorating myocardial function in aortic stenosis, demonstrated by both color Doppler tissue imaging and recently by two-dimensional speckle-tracking echocardiography.
Enhanced sympathetic signaling partially compensates for deteriorating myocardial function [ 9].
Meanwhile unloading (resting) the deteriorating LV has been shown to reverse many cellular and molecular changes in heart failure promoting improvement in myocardial function.
Myocardial function was evaluated using echocardiography.
Increased production of inflammatory cytokines may adversely affect myocardial function.
This study evaluated the effect of intra-myocardial MSC transplantation on myocardial function following MI.
EPO may improve microvascular capacity of ischemic myocardial tissue and could thereby (partially) restore myocardial function.
Myocardial function was evaluated using echocardiography and edge-detection.
Phosphatidylinositol 3-kinase is involved in myocardial function, including contractility.
Acute hypercapnia impairs myocardial function.
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