To study the odds ratio (OR) as measure of treatment effect in the context of mutually exclusive causes of death.
In a strictly hierarchical order, each stillborn or early neonatal death was classified into one of the 13 specific, mutually exclusive causes of death.
Given these two (not necessarily mutually exclusive) causes of the observed rate heterogeneity, it is vital that future studies address the caveats in using the VP1 coding sequence to infer evolutionary rates and history.
Our study highlights that for such species, it may be useful to consider the effects of anthropogenic factors as juxtaposed with the natural phylogeographic structure rather than viewing these as mutually exclusive causes of the observed genetic and distribution patterns.
Several possible non-mutually exclusive causes could be responsible for this: i) dysfunctioning descending aminergic, especially dopaminergic, control [164, 165], ii) malfunctioning hypothalamo-trigeminal control [166], and iii) altered descending opiatergic pain control system [153, 154].
The lack of evidence for local adaptation in our experiment may be explained by several non-mutually exclusive causes.
There are a number of possible and non-mutually exclusive causes of the systematic economies of scale observed in the PANCEA data.
For death cases, where symptoms were contradictory or mutually inconsistent, the cause of death was categorised as indeterminate.
These two mechanisms of action, which are not mutually exclusive, can cause changes promoting abnormal electric activity increase, distorted signal transduction and can eventually lead to cell death [ 16, 23, 24].
