Sentence examples for mutations were explored from inspiring English sources

Incidences of EGFR and KRAS mutations were explored further in a subsequent expansion of the meta-analyses.

Only 5% of patients had a TP53 mutation, although it should be noted that only 12% of known P53 mutations were explored in this study.

The effects of these mutations were explored in studies with human cultured cells stimulated with exogenously added growth factors, which induce a transient pulse of Ras-MAPK activation.

The consequences of LQT3-associated Scn5a+/ΔKPQ mutations were explored using previously developed 1) cellular models of central and peripheral SAN cells (45) and 2) two-dimensional (2-D) tissue models of intact SAN-atrial tissue.

In the current study, molecular mechanism of these resistance causing mutations was explored using molecular docking, molecular dynamics (MD) simulation, binding energy estimation (MMPBSA), free energy decomposition, principle component analysis (PCA) and free energy landscape (FEL) methods.

The likely pathogenicity of the three novel missense mutations was explored by using molecular modeling software.

The key issue of validation of genomic mutations was explored through cross-platform comparison of results obtained from two NGS technologies as well as a mass spectrometry-based method.

In this study, the presence of mutations was explored in a region of 16S-like rRNA gene of E. histolytica, E. dispar and E. moshkovskii by applying riboprinting and SSCP analysis.

Associations between KRAS and BRAF mutation status and clinicopathological factors were explored by Pearson's Chi-square test.

Epigenetic regulation, mRNA expression and mutation screening at the DNA level were explored.

Plausible interactions were explored.