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The mutations vary from a single change in one of the nucleotide bases that constitute the gene to the deletion of the entire gene.
The levels of mutant SOD1 in spinal ventral horns of patients carrying SOD1 mutations vary from equal [15] to around 1% of controls [9].
Mutations vary from point mutations to null expression [136].
Moreover, it is known that the sign and the magnitude of epistasis between mutations vary from one host to another for TEV [ 29].
In the standard account, mutations vary from one population to the next, and are typically multiple and small in effect, with strong convergent selection in similar environmental conditions required to bring about similar phenotypes.
Similar(55)
The mutations varied from person to person but were concentrated in genes known to be involved in brain development.
The frequency of these mutations varies from approximately 30% overall up to 50% in patients with a normal karyotype.
The frequency of these mutations varied from 0 to 8 in the mother child pairs.
The confidence levels of evaluated mutations varied from V2 to V4, and all tested mutations were experimentally confirmed as viable.
The range of changes to localizations of mutations varied from one to five, the highest being for Gtm proteins.
In other Asian countries, the most frequent mutations varied from IVS-I-1 IVS-I-1 IVS-I-1 IVS-I-5 (G>T) in PakIndia [ 16], to −28 (A>G) in Southern China [ 13].
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