Sentence examples for mutations is most from inspiring English sources

In turn, as shown below in figure 4 E, the enrichment of the mutator-associated mutations is most likely due to the benefit that other mutations such as rpoS, mglD, and malT provided at day 40 but this lineage was outcompeted by other members of the same population with different beneficial combinations.

The perceivable differences between the control and experimental groups, inclusion of content addressing timelines, sizes of beaks, and mutations, is most likely attributed to the divergent interaction with the situations of chance tutorial by the groups.

The oncogenic nature of RTKs, addiction of cancers to growth signals and given the clustering of c-CBL, EGFR and MET mutations, it is possible that the transforming effect of c-CBL mutations is most likely a combinatorial effect of the three.

The lower proportional contribution of C>T transitions among late mutations is most likely caused by an increase in the rate of other mutation types because tumor-specific signatures account for much of the variation between early and late mutations.

Because of the problem of PZA susceptibility testing discussed above, the lower percentage of PZA-resistant strains, i.e., 64% and 72%, with pncA mutations is most likely due to false resistance, lack of vigorous retesting to rule out false resistance, the low resistance breakpoint (100 µg/ml PZA in BACTEC or MGIT) used, or the small number of strains analyzed in the study.

Among all somatically mutated genes, CREBBP mutations were most significantly enriched within the earliest inferable progenitor.

Nonsense and frameshift mutations are most common and are associated with severe disease.

Taken together, the evidence suggests that the fitness effects of synonymous mutations are most often either nearly neutral or deleterious and, very infrequently, weakly beneficial.

KRAS mutations were most frequent and identified in 132 (42%) tumors, followed by APC in 79 (25%) and TP53 in 64 (21%) tumors.

The population that carried < 1250 mutations was most often placed in the range of normal proliferation (healthy old SCCs, n = 9), while the majority of SCCs carrying > 1250 mutations showed abnormally long times in culture (diseased old SCCs, n = 7).

Mutations were most commonly found in gastrointestinal samples with 12 of 119 (10%) colorectal and 10 of 100 (10%) gastric neoplasms, respectively, harboring changes.