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Exact(15)
The eight K to R mutations are indicated by asterisks.
The mutations are indicated by boldface, lower-case letters.
Clones with no mutations are indicated.
Locations of domains and genetic mutations are indicated.
All damaging mutations are indicated in Table 2.
Truncation and point mutations are indicated with arrows (bottom).
Similar(45)
Non-synonymous amino acid mutations were indicated to have similar function (yellow) or altered polarity (blue), charge (pink) and aromatic nature (green).
Primer sequences for amplifying FGFR2, deleting 1 kbp in the middle of intron 9, and introducing mutations were indicated in Methods S1.
The spectrum of these mutations is indicated in Figure 2 and Table 2. Sixty percent of the mutations occurred at three sites bp 172 (R58X), bp 238 (R80X) and bp 247 (H83Y)–and were all C→T transitions.
The location of the mutations is indicated by M1 M7.
The fact that the G[8,5-Me]T cross-link lesion was responsible for these mutations was indicated by their absence in the progeny from the control construct.
More suggestions(15)
mutations are noted
transfers are indicated
mutations are incorporated
moves are indicated
transfer are indicated
mutations are summarized
mutations are associated
mutations are seen
mutations are observed
mutations are spread
mutations are mapped
mutations are kept
mutations are replicated
mutations are located
mutations are termed
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