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These disparate phenotypes in mice with hedgehog signaling pathway mutations are incompletely understood.
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The mechanism(s) via which dominant mutations in DNM2 cause myopathy, however, are incompletely understood.
However, phenotypes for mutations affecting axon outgrowth and guidance are incompletely penetrant, indicating redundant mechanisms likely mediate M1 axon extension.
Experimental data regarding the functional effects of 82 of these mutations enabled us to validate the predictions from early in silico models, this information was of particular use during development of our final model and permitted us to identify and learn from mutations which were incompletely assessed by various in silico tools used independently.
Mechanisms of HBV persistence are incompletely understood.
The causative pathophysiologic mechanisms are incompletely understood.
The mechanisms linking the two are incompletely understood.
These drugs are incompletely effective and cause significant morbidity.
However, synaptic mechanisms underlying central sensitization are incompletely known.
However, the effects of FOS on thrombosis are incompletely understood.
The purple bacteria are incompletely oxidizing H2S to elemental sulfur.
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