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Overall, by quantifying synthesis of all published studies of AR, EGFR, and BRCA1, the meta-analysis demonstrated that the expression level of EGFR and the risk of BRCA1 mutation were higher in TNBC compared with non-TNBC and AR expression was downregulated.
In S. pseudintermedius, the MPCs (16 128 μg/ml) and MPC/MIC ratios (16 128) in low-susceptible strains (S7 S10) with one QRDR mutation were higher than those in high-susceptible strains without QRDR mutations (S1 S6; MPC: 2 8 μg/ml and MPC/MIC: 4 16).
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The prevalence of the pfmdr1 86Y mutation was higher after treatment with AQ+SP than after SP, p = 0.002.
The prevalence of KRAS mutation is higher than that in recent molecular studies [ 21, 24].
We find that EGFR is overexpressed and the risk of having BRCA1 mutation is higher in TNBC than non-TNBC.
For example, the K-ras gene mutation is higher in Caucasian NSCLC patients than in Asian NSCLC patients.
The frequency of p.N34S mutation was higher in the group with severe disease (15/164; 9.1 %) and with alcoholic etiology (21/229; 9.2 %).
The recurrence risk is increased after the birth of two affected individuals, presumably because the proportion of germ cells that carry the mutation is higher.
Also, the frequency of mutation was higher in exon 2 than exon 1 (3 cases in exon 2 compared with 1 case for exon 1).
In this study, we demonstrated that frequency of KRAS mutation was higher in LST-G than in LST-NG or polypid tumors.
The proportion of available full-text articles with at least one mutation is higher than in the case of the abstracts.
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CEO of Professional Science Editing for Scientists @ prosciediting.com