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Exact(22)
K-RAS mutational status (wild type vs. mutation) was determined on the purified DNA obtained for the MSI analysis (above) using the Therascreen K-RAS mutation qPCR kit (Qiagen/DxS, Manchester, UK) according to manufacturer's instructions.
Presence of the GSTM1 mutation was determined through blood samples.
The factor V Leiden mutation was determined by direct sequencing and the MTHFR genotype by a PCR-based RFLP method.
The LSE1 mutation was determined to be caused by the disruption of a gene encoding α-glucan, water dikinase, OsGWD1 (Os06g0498400; RAP_DB; http://rapdb.dna.affrc.go.jp/).jp/
The amino acid at codon 129 in coupling with the mutation was determined by cloning.
The status of the JAK2 V617F mutation was determined by a DNA tetra-primer ARMS assay as previously described [37].
Similar(38)
The location of a point mutation is determined randomly.
Sex and Ta mutation were determined by PCR-based genotyping [13].
Although the mutated chromosome is identified by a significant shift in allele distribution, the final candidate region containing the mutation is determined by identifying the position of the crossover event in each recombinant progeny.
The presence or absence of mutations and the clonality of each mutation were determined for each marker.
In the blood samples and buccal swabs prothrombotic mutations including the Factor V Leiden (G1691A) mutation were determined.
More suggestions(15)
transfer was determined
move was determined
mutation was described
mutation was detected
mutation was repeated
mutation was labeled
mutation were determined
mutation was genotyped
mutation was created
mutation was backcrossed
mutation was introduced
mutation was expected
mutation was termed
mutation was named
mutation was identified
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