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Interestingly, a concurrent KRAS G12D mutation was also detected in this patient.

A single nonsense mutation was also found among MLK3 mutated MSI cases (Table  2).

The DNAJB2 c.352+1G>A mutation was also detected in three additional families.

The L194P mutation was also spontaneously selected during egg propagation of E611/PR8 7 1 recombinant virus.

Using next-generation sequencing (NGS), we found that this A948G mutation was also stable up to seven passages of DN-2 on Vero cells (Table 1).

The C207S mutation was also generated in cloned human sulfite oxidase.

The mutation was also produced in the VT1 operon, and mutant holotoxin was purified to homogeneity.

The R83C mutation was also demonstrated in one homozygous and five heterogenous GSD type 1a patients, indicating that type 1aSP is a misclassification of GSD type 1a.

This mutation was also found in a Dutch family that shares the same disease haplotype as the Antillean families with this mutation.

This mutation was also found in QH-like viruses.

An antiphospholipid syndrome was ruled out and the JAK2-mutation was also not detected.