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Exact(5)
In ancestral, control, and phage-exposed populations, mean abundance varied widely and b was not significantly different from 2. Evolutionary factors (mutation rate, selection) and ecological factors (abiotic, biotic) jointly influenced b.
This time scale is determined by a "scaling factor" that is a function of the mutation rate, selection coefficient, and effective population size.
As strand or frame should not influence slippage mutation rate, selection seems to modulate the frequency of fixation of LCRs of different composition.
By comparing different substitutions rates within the same analytical framework, we can examine the reasons for the proposed acceleration in rates in parasitic plant taxa, by teasing apart the patterns due to mutation rate, selection and population processes.
Numerous processes are known to determine the frequency of disease mutations, namely: mutation rate, selection, genetic drift, and founder effects and sufficient data does not currently exist to determine the role, if any, of these factors.
Similar(55)
As a measure of evolutionary foresight, we analyze the probability that the double-mutant v fixes as a function of the relevant mutation rates, selection coefficients, and population size.
These challenges include accounting for evolutionary complexities such as changing mutation rates, selection, reassortment, and recombination, as well as epidemiological complexities such as stochastic population dynamics, host population structure, and different patterns at the within-host and between-host scales.
Theoretically, it is expected that effective population size will be the major factor determining gene diversity even at a locus under balancing selection, if the mutation rate and selection coefficient are constant [ 5- 7].
He found that pollution could both decrease and increase genetic variation in populations (defined as genetic diversity or mean number of alleles per locus) through genetic drift and directional selection on the one hand and increased mutation rate and selection for heterozygotes on the other hand.
Although speculative, a smaller gene complement could permit larger fluctuations in mutation rate because selection against a mutator allele would be proportional to its effect on the rate of deleterious mutations per genome, not per nucleotide (Smith and Keeling 2015).
For simplicity, we assumed that all of the selected loci have the same mutation rate and selection coefficient.
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