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A yeast model of this mutation provided a strong indication that the disease was indeed caused by defects in mitochondrial protein import (Mehawej et al., 2014).
The identification of this mutation provided a biochemical and physiological explanation for the origin of PSS and the occurrence of PSE meat in pigs.
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Nevertheless, the functional analysis of this mutation provided an important and interesting novel insight in the biological significance of the β′-site processing of APP, which appeared quantitatively much more important for normal APP metabolism than previously thought.
This mutation provides a mouse model for panhypopituitarism.
If a mutation provides a better adaptation, as Dr Cochran thinks these disease genes did in financiers, it will spread.
It is shown that decimal coding, processed using ranking, elitism and mutation, provides a satisfactory computational basis and offers certain advantages over binary versions.
The improved specificity of MGB probes for single base pair mismatches such as the +93 uidA mutation provides a novel approach towards rapid identification of E. coli O157 H7.
Gene duplication followed by the fixation of a mutation providing a different function, is one of the major sources to create genetic novelty [1].
Connecting viral variants that differ in one mutation provides a natural framework to organize the heterogeneous population of viral variants.
These similar values indicate that the T6′S mutation provides a constant multiplicative factor on other mutations and should not affect measurement of binding site interactions.
Ongoing mutation provides a substrate on which selection operates, with aberrations yielding increased fitness leading to an increasing proportion of affected cells and their progeny [ 2].
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