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XDR-TB, an even more extreme and deadly mutation, has been reported in 92 countries.
The site of the mutation has been shown in italics in the above three primers.
In addition, mutation has been introduced in QPSO for avoiding the premature convergence.
Recently, EGFR mutation has been shown to correlate with responsiveness to tyrosine kinase inhibitors (TKI).
The high incidence of K-Ras mutation has been reported in pancreatic, colon, and lung carcinomas.
Recently, heterozygosity for pax6 mutation has been reported in some individuals with glucose intolerance and aniridia.
SOD1 G93D mutation has been described in amyotrophic lateral sclerosis (ALS) patients with slowly progressive disease.
The V94M mutation has been associated with a very severe form of type III galactosemia.
The M694V mutation has been shown to be a strong risk factor of developing amyloidosis in different ethnic groups [17].
This mutation has been shown to be an activating mutation both experimentally [35] and computationally [36].
The T58A mutation has been identified in several cases of Burkitt's lymphomas.
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