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The glp-4(bn2) mutation renders the strain incapable of producing progeny at 25°C [11] and the sek-1 km4) sek-1 km4causes enhanced sensitivity to various pathogens [12], thereby decreasing assay timutation
Similar to the related Semliki Forest virus, in which a homologous mutation causes enhanced membrane fusion capacity in insect cells, the virus could have an advantage in insects or even in humans (22, 24 ).
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We expected that mutations in genes that promoted miRNA-mediated silencing of the eGFP reporter would cause enhanced fluorescence within mitotic eye clones.
These gene knockdowns caused enhanced neurodegeneration.
128, 129 Mutation of Nr2e3 causes enhanced S cone syndrome (ESCS), 130 a retinal degenerative disease in humans that results in an abundance of short-wavelength sensitively cones (S cones) at the expense of rod photoreceptors.
Indeed, recent description of an apoptosis-enhancing cytochrome c mutation causes thrombocytopenia in individuals carrying it.
Our observations that the MRH2 mutation causes cortical MT to be randomly oriented and fragmented and greatly enhances the CA-rop2-activated disruption of MT organization (Figure 4) suggest that MRH2 is important for stabilizing MT or maintaining MT orientation.
The cpr1 (constitutive expresser of PR1) mutation causes constitutively high SA levels, increased PR1 expression in the absence of pathogen, and thus enhanced disease resistance [ 16].
Both the sterile lemma elongation and the floral organ number increase phenotype are enhanced by the mutation of an independent gene SMALL DEGENERATIVE PALEA1 (SDP1), whose single mutation causes reduced palea size.
This mutation causes both FJHN and MCKD.
The significance of the NR2E3 gene was recognized following the discovery that mutations in this gene cause enhanced S-cone syndrome (ESCS), with 'gain-of-S-cone function'.
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