Sentence examples for mutants with wild from inspiring English sources
Shake flask experiments were carried to compare the mutants with wild strain for β-glucan production.
The study is based on genetic rescue of endophilin-A (endoA) null mutants with wild type or mutated endoA transgenes.
To test whether this is applicable to pathogenic E. coli EDL933, we compared the growth of rpoS mutants with wild type EDL933 grown in LB.
Transgenic mutants with wild type coding sequence complemented the salt tolerance phenotype and were similar to wild type plants when grown on MS medium supplemented with 125 mM NaCl for salt stress (Additional file 2: Figure S1A).
Moreover, all patch clamp experiments directly compare mutant responses (cpk, snrk2 and pp2c mutants) with wild type responses and show strong and clear differences to the wildtype response that correlate with genotype-blinded stomatal movement analyses that do not use protoplasts here.
We compared one of each subclass of mutants with wild-type parasites; mutants 13P and B7 were chosen as representative mutants for the two different subclasses.
To test for putative parent-of-origin effects, we reciprocally crossed pbrp2-1 mutants with wild-type plants and examined endosperm development in F1 seeds.
Further, comparisons of double p53−/−shh−/− mutants with wild-type, and shh−/− mutant embryos identified p53-mediated inhibition of retinal progenitor proliferation, cell-cycle exit and differentiation of amacrine cells and photoreceptors.
Comparing the SOD mutants with wild-type animals, we further show that oxidative damage levels in the mtDNA of SOD mutants are not significantly different from those in wild-type animals, i.e. even the total loss of mitochondrial SOD did not significantly increase oxidative damage to mtDNA.
We therefore tested whether reduction of galactinol contents resulted in enhanced ROS sensitivity in atips1 mutants, with wild-type and atips1 plants cultivated under SD on MS for 12 days and then transferred to MS medium supplemented with various drugs known to induce oxidative stress (see Figure S6 for details).
The optimality model was tested in phage λ and T4 by competing lysis mutants with wild-type phages.
