Sentence examples for mutants with age from inspiring English sources

However, Leans and Controls showed few immunopositive cells for CD11b/TNF α in comparison to Mutants with age– 4 e)).

All experiments were conducted in synchronous young adult animals at a temperature of 20 ± 1°C to avoid the paralysis that occurs in unc-52 and unc-112 mutants with age and also following shifting to the nonpermissive 25°C (both strains display uncoordinated movement and sarcomere defects at young adulthood at the permissive 20°C).

Figures 4(a)– 4 e) depict dual immunofluoresence for CD11b/TNF α (RFU per unit area with reference to isotype control)), that is, relative percentage of macrophages/mast cells/inflammatory protein in situ which was significantly upregulated in Mutants with age.

Gene expression differences were determined by one-way, error-weighted anova on ratio data by comparing Aβ1 42 and Arctic mutants with age-matched Aβ1 40 controls (factor = genotype, n = 4 per group).

The number of PH3-positive cells in both mutants decreased with age.

CD11b studied here is a macrophage marker that is highly expressed as a surface marker of activation on resting monocytes along with others such as CD11c and HLADR [ 32] which correlates with the increased macrophage and T-cell activation among Mutants and with age.

Children recognised both sets of mutants with different age groups having different recognition levels.

No significant difference in this parameter was documented between the ACHE mutants, with both age groups being similar to young wild-type zebrafish.

In the present study we have compared homozygous obese rats (WNIN/Ob (Mutants) with their age matched homozygous lean (WNIN/Ob littermates (Leans) and parental (WNIN) controls (Controls) for all the parameters (islet cell architecture, inflammation, and islet cell functions) studied in in situ (tissue) and in vitro (primary islet cell cultures).

For these mutations, a very good correlation was found between the super-linearly increasing birth data (4) and the mutation frequency measured in sperm (23), suggesting that the PAE observed in the population is correlated with a higher number of mutant sperm with age, driven by a selective process during spermatogenesis.

The identification from the 1980s onwards of many C. elegans mutants with altered aging rate [ 1] led to optimism that discovery of gene products of aging control genes would reveal the mechanisms of aging in this organism.