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Stress-induced HRF and DNA methylation were impaired in dcl2 and dcl3 deficiency mutants, while in dcl2 mutants, only stress-induced stress tolerance was impaired.
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Such situation could be explained as a chance event that the embryo could end up into any one of the several potential crypts in the Rbpj mutant uteri (while in normal uteri, there is only one crypt with its axis parallel to uterine M-AM direction), establishing an embryonic axis that is dependent on the direction of the crypt that it accommodates with.
Figure 2c showed higher nitrate content in the nlp7 mutants while lower in the chl1 plants than that in WT (Fig. 2c).
The HIM-3-dependent linkages are not mediated by an SC-like structure, given that disrupting the CR protein SYP-1 only partially weakened SCC in spo-11 rec-8 double mutants, while mutations in him-3 severely abrogated SCC.
The double cpc etc simulated mutant gives rise to few clusters, yet not as many as the try simulated mutant, while in the actual cpc etc mutant trichome clusters are extremely rare.
The production of tensidol B on CYA was absent in the Δ laeA mutant, while in the D15 mutant, which contains a point mutation in the laeA gene, tensidol B was still produced.
In the proband, direct sequencing of the NOTCH3 cDNA obtained by retrotranscription of the residual mRNA detected only the 2898A allele (mutant), while in the heterozygous parents, the C2898 allele (wild-type) appeared to be predominant (Fig 2B).
The humeral deltoid tuberosity was significantly reduced in size in the Myf5 nlacZ/nlacZ MyoD −/− mutants while a change in shape but not in size was found in the humeral tuberosities of the Pax3 Sp/Sp mutants.
A synergistic relationship between tej and tap for piRNA production is also suggested by the mis-localization of Piwi from the nucleus in germline cells of double mutants, while Piwi stayed in the nucleus of germline cells in both single mutants.
Among the mutants, the highest levels of ATL9 induction were detected in the ein2-5 and npr1-1 mutants, while ATL9 levels in sid2-1 and jar-1 were the lowest (Figure 2A).
JM participated in the construction of the mutants, while HL helped in the statistical analysis of the data.
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