Sentence examples for mutants when subjected from inspiring English sources

The exacerbated decrease in the bacterial fitness of prfA* mutants when subjected to two different stress conditions suggested that a general response related to stress tolerance may be compromised by constitutive activation of PrfA.

The double mutants did not exhibit significant differences in the root length and fresh weight compared to the single mutants when subjected to salt and osmotic stress (Additional file 1: Table S1).

Transgene-rescued EPOR null mutant mice when subjected to IRI showed a larger infarct size and reduced left ventricular (LV) function compared with wild-type mice with intact EPOR [29].

Studies in yeast showed that a truncated version of AtHMA4, Athma4Δ714 1172 (lacking the cytoplasmic C-terminal region) conferred greater Zn tolerance than full-length AtHMA4 to the Zn-sensitive mutant zrc1 cot1 when subjected to elevated Zn and greater Cd tolerance to wt yeast [11].

Transgene-rescued EPOR null mutant mice when subjected to IRI showed a larger infarct size and reduced left ventricular (LV) function compared with wild-type mice with intact EPOR [ 29].

The abscisic acid oversensitive cbp20 and era1 Arabidopsis mutants retain more water when subjected to limited water supply due to restricted gas exchange, resulting in improved drought tolerance.

The tgs1 deletion mutant failed to accumulate TG when subjected to multiple-stress condition, while complementation of this mutant with tgs1 restored the ability to store TG under the same multiple-stress treatment (Fig. 10).

According to a previous study, some phosphoethanolamine N-methyltransferase mutants exhibited pale green leaf color when subjected to high salinity [ 14], which may indicate a decrease in leaf chlorophyll levels.

However, we observed that rict-1 mutants provided with OP50 lived much longer when subjected to DR in a liquid-feeding regimen, arguing against this idea (Fig. 5A,B, Tables S10 and S11, Supporting information).

At rest, null mutants do not differ in their heart rates from wild-type; however, when subjected to either physical or mental stress, the mean heart rate of the null mutants is significantly lower.

Taken together, these results indicate that mutant hyperactive FGFR2 signaling generally has a protective effect against Myc-dependent apoptosis and that MEFs oncogenically transformed by the combination of FGFR2K569E plus c-Myc appear to become highly addicted to the prosurvival activities of the mutant receptor and, as a result, highly susceptible to apoptosis when subjected to FGFR inhibition.