Shimono et al. [ 4] showed that an mce1 mutant was hypervirulent when inoculated intravenously into BALB/c mice.
The ΔBAR1 mutants were non-pathogenic when inoculated onto wheat heads, and their in vitro growth also was severely perturbed.
Interestingly, although the Ybt– mutant was highly attenuated when inoculated intradermally by needle, fleabites from fleas infected with this mutant produced terminal disease in two of ten mice, at 3 and 6 days post-challenge (Figure 1 and Table 1).
Both wild type and sod1Δ mutant cells grew well when inoculated into zinc-replete medium (LZM + 1000 µM ZnCl2) (Fig. 2A).
The lsd1 mutant exhibits runaway cell death when inoculated with avirulent bacterial pathogens, suggesting that AtLSD1 (AGisat4g20380) is a negative regulator of PCD [7].
Nevertheless, ΔBAR1 mutants did not trigger any symptoms when inoculated onto wheat heads.
Compared to cells inoculated with the C. jejuni wild-type strain, cells produced significantly less activated Rac1 when inoculated with the flpA mutant or the flpA mutant synthesizing the FlpA Δ158 164 or FlpA P160A D161C proteins.
However, when inoculated with aap bra mutants, beans are less severely nitrogen starved than peas.
The absence of nodulation in both leucaena and siratro when inoculated with the nodD1 mutant was confirmed.
In the case of the plant pathogen Ralstonia solanacearum, for example, a chemotactic mutant is not able to colonize its host when inoculated in the soil, whereas it retains full pathogenicity when infiltrated directly in the plant tissues [94].
