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All these suspected mutants were sucrose-sensitive.
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Results indicate that docs1 mutants were less sensitive to gravistimulation than control plants.
All the other mutants were EndoH sensitive (Fig. 2 A).
Resolved mutants were isolated on sucrose and confirmed for loss of the plasmid backbone by lack of growth on tetracycline.
Both mutants were unaffected by sucrose (Fig. 1).
Seventy-six deletion mutants were identified as vanillin-sensitive mutants.
It is likely that ΔscrA and ΔscrAΔscrB use the complex carbon source for growth even in the presence of high concentration of sucrose since these mutants were unable of transporting sucrose into cell.
The pmt3-K14R and pmt3-K14R,K30R mutares are both sensitive to HU (5 mM), while pmt3-K30R is very slightly sensitive to HU.
Many DNA-repair gene mutants are very sensitive to CPT.
Double halA− slt− mutants are extremely sensitive to Mg2+.
However, the compound mutant shows resistance to ponatinib, while the two mutants are individually sensitive.
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