These mutants were found to be cross resistant to FTC.
In addition, a few gene product alterations specific for each of the multi-step mutants were found.
Rather, multiple W-to-G substitutions were required to inactivate Cef1p, and many of the substitution mutants were found to confer temperature sensitivity.
The mutants were found to be less than half as responsive as their wild-type conspecifics to the highest modafinil dose (40 mg/kg) (Stone et al, 2002a).
Five tall mutants were found in DongnongV7 variety, and the average height of the mutants was 31% taller than that of the control.
Most mutants were found to exhibit vastly improved Vmax values and display an increase in high cell density sugar consumption rates.
In contrast, all the mutants were found to have altered NADH ferricyanide reductase (NADH FR) activity with mutations affecting both kcat and KNADHm, which decreased and increased, respectively.
Both mutants were found to be largely helical as per design, and based on thermal denaturation experiments, were substantially more stable than the MB-1 parent molecule.
The tga3-1 mutants were found to be defective in basal pathogen resistance, whereas induced resistance was unaffected.
A total of 248 mutants were found to be completely missing in the 2nd round screen.
These Escape mutants were found to have a common mutation at Ser 155.
