Sentence examples for mutants we set from inspiring English sources

In the light of the reduced curvature in JAK/STAT pathway mutants, we set out to identify the reason for this phenotype.

To simulate the dynamics of mutants, we set the mRNA synthesis rates for two of the three IкB isoforms and A20 to zero.

In addressing the molecular basis of the neuropathogenesis of PEX13 brain mutants, we set aside the longstanding, but ill-supported, view of a disturbance of peroxisomal intermediary metabolism being the primary cause of ZS pathogenesis.

Although the analysis of SVZ neurogenesis was not reported for Tsc2c/− /hGFAP2- Cre+ mutant mice, we set out to assess whether mTORC1 hyperactivation in RGCs would result in SVZ alterations, as previously reported in Tsc1c/− /Emx1- Cre+ mice (Magri et al., 2011).

Because of the expression differences in the Δ wc-1 or Δ wc-2 mutants, we evaluated whether genes sets were regulated (directly or indirectly) by the WCC-complex (similar regulation in both mutants) or regulated (directly or indirectly) by WC-1 or WC-2 individually, but not by the WCC (Additional file 5 Dataset 4).

In the training set of mutants, we denote the subset of instances seeing increase and decrease in solubility by I and D, respectively.

To find the sites of the lesions in the sets of mutants, we pooled several genomic DNAs and amplified aliquots using 1.25U Optimase Polymerase (Transgenomic, Inc).

Using programmed cell death mutants and pathogen infection we set out to investigate the role of stomata in biotic stress.

To test the robustness of gene expression in this set of hormone mutants, we compared two different growth conditions.

This set of proteins is independent of the set of 137 solubility mutants we have assembled, which is described below.

Interestingly, for the 506-set corresponding to genes deregulated in both mutants, we observed a higher enrichment in responses to abiotic and biotic stress stimuli compared to the two mutants sets (Fig. 6A).