Sentence examples for mutants we observe from inspiring English sources

With the exception of the PC-ΔPP2B mutants, we observe that despite different pathways being affected by those mutations, whether it is GC excitability, PF-PC plasticity or MLI-PC inhibition, at the level of Purkinje cell activity, the outcome is a net reduction in depth of the SS modulation.

In dFatp mutants, we observe abnormally high myocardial autophagy.

In fact, in PP2A mutants, we observe an enhanced fusion growth defect, suggesting that disruption of PP2A causes cellular stress, which elicits, rather than compromises, Pol III repression.

Within the library of transcription factor deletion mutants, we observe significant differences in the sets of genetic interactions identified by the fitness- and sensitivity-based approaches.

When the Rad51-dependent strand invasion step of HR is inhibited in spn-A mutants, we observe a significant increase in SSA rather than by NHEJ.

In ethanol-treated mutants, we observe a decrease in the levels of activated eIF4B and an increase in levels of activated AKT, suggesting that ethanol impairs growth signaling at the level of mTOR or below.

In ap1b1 mutants we observed that although the overall development of the inner ear and lateral-line organ appeared normal, the sensory epithelium showed progressive signs of degeneration.

Along with enlarged presynaptic ribbons in ca(V 1.3a mutants, we observed a profound loss of juxtaposition between presynaptic and postsynaptic components.

In tmie zebrafish mutants, we observed that GFP-tagged Tmc1 and Tmc2b, which are subunits of the MET channel, fail to target to the hair bundle.

In Afh mutants we observed chronic alteration of the sleep physiology (i.e., NREM fragmentation and delta power reduction), confirmed by an abnormal Process S. The sleep changes in baseline were followed by a prolonged delta power decrease after sleep deprivation (Fig. 7b).

To measure PEV in Slbp mutants, we observed the extent of variegation of the wm4 inversion.