Sentence examples for mutants we find from inspiring English sources

For both wild-type PDZ domains and single point mutants, we find that 70 80% of the most frequently observed amino acids by phage display are predicted within the top five ranked amino acids.

Through the analysis of a series of E1A mutants, we find that sequences in conserved region 1 (CR1) and conserved region 2 (CR2) are important for dissociation of the E2F complex, whereas amino-terminal sequences are not required.

Repeating the analysis for the dopamine mutants, we find the interval distributions statistically identical (Table 1).

In these mutants, we find multiple mesenchymally-related defects that are remarkably reminiscent of a double knock out of Pitx1 and Pitx2 genes [19].

In unc-116 (kinesin-1/kinesin heavy chain) mutants, we find that dendritic MT polarity is completely reversed and adopts an axonal-like plus-end-out organization.

Here, however, by comparing multiple AID NES mutants, we find that – beyond a requirement for threshold Crm1 binding – there is little correlation between CSR and Crm1 binding affinity.

Examining 48 additional tyr a chromosomes in albino mutants, we found evidence of historical recombination between thrsp and tyr in only 12 cases.

When applying the same analysis to several barnase mutants, we found that single mutations may drastically change the free-energy landscape and may significantly alter the population of the two minima.

Using a series of ERalpha mutants, we found that helix 12 was not required for the binding of CoRNR box peptides, whereas disruption of helixes 3 and 5 had a marked effect on peptide binding.

When we tested the cleavage resistance of V5-MKK7cr_45 V5-MKK7cr_77r_77 mutants, we found both of the mutants were resistant to the cleavage in CHO K1 cells (Figure 2F).

By examining the morphology of motor neurons in the mutants, we found that some VD neuron axons showed defective morphology in several mutants (Figure 1C and Table 2).