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Through the analysis of T-DNA insertion mutants, we also found that most of the other subunits are dispensable.
Using cryb mutants we also detected that CRY is not only the circadian photoreceptor.
In notch1a/desth35b mutants, we also detected an increase in foxi3a expression within the IC domain (Figure 4G; Table 1).
To further evaluate replication abnormalities in swi3 mutants, we also monitored the formation of Rad22-YFP DNA repair foci in the absence of genotoxic agents.
To gain further evidence of the occurrence of a reeler-like phenotype in Pafah1b1+/− Apoer2−/− double mutants, we also examined the anatomy of hippocampal structures (Fig. 6).
Given that the abundance of dMfn was increased in PINK1 and parkin mutants, we also tested the effects of PINK1 and Parkin overexpression on dMfn abundance.
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Because expression of alsSD was increased in a biofilm but decreased in a sarA mutant, we also generated a plasmid construct that allowed expression of alsSD in a sarA mutant.
In an effort to better relate the combination of the Wap-p53R172H and Neu transgenes with our Rb1 mutant, we also investigated metastasis in Neu; Rb1+/+, Neu; Rb1ΔL/+, and Neu; Rb1ΔL/ΔL female mice.
To identify the underlying mechanisms for the observed compromised resistance in the aos-hpl mutant, we also examined the levels of camalexin, the main phytoalexin in Arabidopsis shown to inhibit growth of some Botrytis cinerea isolates [31].
In addition to the Akt1-DD mutant, we also used two other constitutively active mutants of Akt.
For each mutant, we also asked which genes have different overall patterns of expression compared with the wild type across the time course.
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