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Nrp1 cytoΔ /Δ mutants undergo normal vasculogenesis and angiogenesis, demonstrating that membrane-anchored extracellular NRP1 is sufficient for both processes.
Unlike sma-1 mutants, sma-2 and sma-2 mutants undergo normal elongation and have normal pharyngeal morphology, but are smaller than wild type due to reduced postemutantsc growth of cells.
We also blocked Nodal signaling in M ybx1 mutants by injecting lefty1 inhibitor RNA and find that the injected mutants undergo normal cytokinesis and gastrulation, unlike mutant embryos injected with control lacZ RNA.
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However, when a hypomorphic allele of e-cadherin mutant, habrk3, is maintained at 31°C, habrk3 mutant embryos undergo normal gastrulation (Shimizu et al., 2005).
We also found that AtRaptor1A AtRaptor1B double mutant homozygotes undergo normal seedling development, but exhibit only minimal post-embryonic meristem activity.
Parasite transmission experiments using these mosquitoes resulted in infection of mice with both WT and Δ shlp1 lines, strongly suggesting that the mutant parasites undergo normal exo- and intraerythrocytic proliferation, but oocyst development in vivo is ablated.
The HSC activity was also normal at a later stage (E14), when HSC expansion occurs in the FL, indicating that α IIb mutant HSCs can undergo normal expansion.
Here we present the analysis of seven insertional mutants that do not undergo normal bradyzoite differentiation.
We present the analysis of seven novel T. gondii insertional mutants that do not undergo normal differentiation to bradyzoites.
The WT and mutant fibers appeared to undergo normal initiation as visualized on the DOA with no apparent differences in the distribution or density of fiber initials on the ovule surfaces.
Here we describe the isolation of seven mutants that are unable to undergo normal bradyzoite differentiation.
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