Sentence examples for mutants under low from inspiring English sources

In this study, the tcd10 mutants, under low temperatures (20 °C), had the severe affected transcript-levels of most genes for Chl biosynthesis, photosynthesis and chloroplast development (Fig. 6), whereas the down-regulated genes at low temperatures could be recovered to normal level or even higher WT levels at high temperature (32 °C) (Fig. 7).

The initial genetic and physiological analyses revealed growth deficiency of sos mutants under low K+ conditions, which leads to an assumption that sos mutant loci are essential components for K+ acquisition and signal transduction during salinity stress (Wu et al. [1996]; Zhu et al. [1998]).

To test whether Delg might mediate the reduction in mitochondrial abundance upon low-yeast starvation, we grew delg mutants under low yeast and labeled mitochondria using mitoGFP.

Consistently, survival of rpoS mutants under low pH, oxidative stress, and heat exposure was severely impaired in comparison with wild type EDL933 strain.

Moreover, the heading-date of mutant under low temperature was 11 days earlier under LD conditions than under SD conditions.

Furthermore, LNs decreased under LD and low temperature treatments in the mutant, in contrast to the wild type LNs, which increased with the change of day length from SD to LD, whether under the high temperature or low temperature treatments, indicating that LD conditions promoted the heading-date of the mutant under low temperature treatment.

The SD promotion rate for the wild type was 16.5%, but the SD promotion rate for the mutant was negative, −19.4% (Table 1), indicating that photoperiod had a negative effect on the heading-date of the mutant under low temperature conditions.

The CBL10-overexpressing lines exhibited a K+-sensitive phenotype, like that of the akt1 mutant under low K+ conditions.

Genes involved in mitochondrial OXPHOS activity, including RFeSP and Blw, showed similar reduced expression in the delg mutant or under low yeast nutrition.

In our previous work [ 16], cDNA microarray was used to determine the expression profile of a Y. pestis phoP null mutant grown under low Mg2+ conditions, enabling the genome-wide screening for PhoP-dependent genes in Y. pestis.

As the selection of p53-defective cells has been described as a consequence of cyclic hypoxia and Wt p53 undergoes conformational change in to mutant form under low oxygen concentration, we asked if re-oxygenation at normobaric pressure could trans-activate p53 and its downstream genes in regressing of hypoxic tumor.