Sentence examples for mutants treated with from inspiring English sources

In contrast, there were no significant differences in the expression of OsAS2 and Actin1 between the shoot basal portions of wild type and gs1 2 mutants treated with or without NH4+ (Fig. 4b, c).

Note that these single mutants treated with control RNAi (empty vector) displayed a superficially wild-type (non-Muv) vulval morphology.

We found reproducible changes in fatty acid composition of wild type, fat-6;fat-7, and sbp-1 mutants treated with nhr-64RNAi (Figure 3).

In E. coli hydroxiperoxidase mutants treated with micromolecular levels of hydrogen peroxide, Fur is unable to control the free iron levels since oxidation of Fe2+ to Fe3+ by hydrogen peroxide inactivates its repressor activity.

In contrast to the mutants treated with TM at E10.5, Hh-GOF mutants did not display an omphalocele even with the higher dose of TM treatment (4 mg/40 g bw) at E12.5 (Fig. 5E).

We found that the decrease in 18∶0 correlates with improved growth and increased fat storage in the fat-6;fat-7 and sbp-1 mutants treated with nhr-64 RNAi). nhr-64 RNAi

Mutant lacking the gene for GSH synthesis (gsh1) exposed to AgNPs and AgNO3 obtained roughly the same OD values compared to control, except the last two highest concentrations that the values decreased in mutant treated with AgNO3.

Briefly, root tips or basal roots of WT and the mutant treated with 20 μM Al or 5 μM Cd for 6 h were put in Milipore Ultrafree-MC Centrifugal filter units.

From 3.0 mg/L AgNPs, the absorbance values were lower than control, whereas at 12.5- and 25 mg/L AgNPs, the absorbance values were similar to the mutant treated with AgNO3.

In case of sod2 mutant (lacking of the mitochondrial superoxide dismutase gene), absorbance values decreased with increasing concentration of AgNO3, while in mutant treated with AgNPs the values were similar to control and began to decline at 6.25 mg/L AgNPs and remained the same when treated with higher concentration.

We next examined checkpoint responses in cells expressing the dpb11Δ C mutant treated with phleomycin.