Sentence examples for mutants to explain from inspiring English sources

Mathematical models have also been compared with mutants to explain the multi-cellular arrangement of Arabidopsis root hair and non hair cells [50].

Such variability in molecular mechanisms observed in the hClC-1 mutants may help to explain the different clinical and neurophysiologic manifestations of each ClCN1 mutation.

The reason for the arbutin resistance of the 4 other mutants is difficult to explain and is probably not related to OPGs suggesting that arbutin affects several different cellular processes.

Analysis of the triple mutant structure and the accompanying molecular dynamics simulations reveal that the constitutively open cytoplasmic side of the triple mutant may help to explain both its ability to pump protons (reprotonate the Schiff base) in the absence of the internal proton donor Asp96 and its inability to pump against a physiologically relevant negative membrane potential.

The reduction in the S unit content can easily be explained for the ccr1, f5h1, and comt mutants, but the increase in S units in the c4h and 4cl1 mutants is more difficult to explain from the linear pathway presented in Figure  1.

The CPR gain-of-function mutants were more difficult to explain, but Miller and colleagues suggest that, at least in the case of the Q153R mutant, electron transfer rates to the CYP may be affected by the close proximity of glutamine residue 153 to the FMN moiety.

This approach can be used also to explain mutants shapes.

Complementing dpolv nonsense mutants, are also difficult to explain by cis alternative splicing.

However, because the nodD1 mutant is unable to nodulate leucaena, other LCOs not secreted by this mutant must be important to explain its symbiotic phenotype.

However, the reason for the increase in sensitivity of the ΔruvABC mutant is not easy to explain, as no Holliday junction nuclease is known to act in conjunction with RecG and the pulsed field gel electrophoresis presented here shows that no cleavage-ligation of Holliday junctions can be detected in the presence of RecG and absence of RuvABC.

Several models have been suggested to explain mutant p53 gain of function.