Sentence examples for mutants shows that from inspiring English sources
However, morpholino-mediated knockdown of Tie-1 function in tie-2 zebrafish mutants shows that combined loss of Tie-1 and Tie-2 function results in defects of myocardial-endocardial contact.
Lys12 is located in a lysine-rich region of the protein but analysis of a set of Lys to Met mutants shows that it plays a unique role in the denatured state.
Experimental analysis of the mutants shows that the C terminus of the molecule (comprising the last and edge β-strand) is the major contributor to amyloid fibril formation, in good agreement with theoretical predictions.
Protein engineering and kinetic (ϕF-value) analysis of a series of Uβ4 mutants shows that the hairpin extension of Uβ4 is also significantly populated in the transition state (ϕF-values >0.7) and has the effect of templating the formation of native contacts in the folding nucleus of ubiquitin.
Steady-state kinetics of methionine mutants shows that ferrocytochrome c oxidation is <2% of wild type levels without affecting the second order rate constant of first phase of Compound I formation, while the activity toward a small molecule substrate, guaiacol or iodide, increases.
The high frequency of mitotic recombination within the rDNA cluster in topoisomerase mutants shows that both TOP1 and TOP2 are required for suppression of recombination in this region of the genome.
As analysis of the akin10 and akin10/11 mutants showed that bZIP63 is phosphorylated by more than just AKIN10, as phosphorylation was not abolished completely in these mutants.
Studies with mutants showed that ChrR protects against chromate toxicity; this is possibly because it preempts chromate reduction by the cellular one-electron reducers, thereby minimizing ROS generation.
An immunoblot analysis of HEK293T cells transiently overexpressing the three mutants showed that they could be expressed at similar levels (Fig. 5B).
39, 248 259] with engineered Salmonella mutants showed that deletion of SopE attenuated the pathogen's ability to deplete host-cell InsP5 and remodel the cytoskeleton.
Biochemical data on RF3 mutants show that a surface region involving domains II and III is important for distinct steps in the action cycle of RF3.
