Sentence examples for mutants shared by from inspiring English sources

Using the datasets available from three representative screening approaches [ 5, 8, 10], we started with pairwise comparisons among the three datasets and characterized the number of common pairs of array and query mutants shared by the datasets (Table 1), as well as the distribution of the known pairs of positive and negative interactions into the data intersections (Table 2).

Both YnaI-Ttβ and YnaIF209A mutants shared a similar reversal potential with YnaI.

Only the non-missing mutant pairs shared by the datasets were included in the comparative analyses.

The congruence between the dataset pairs was evaluated by calculating the Pearson and Spearman correlations across those mutant pairs shared by both datasets.

The values above and below the diagonal give the Pearson and Spearman correlation, respectively, as calculated over the non-missing mutant pairs shared by each dataset pair.

It is worth noting that H4-R36A appears to be generally a "weaker" mutant than the H3-L61W mutant because although the two mutants share similar phenotypes, those displayed by H4-R36A cells are generally milder and are more easily suppressed by the Spt16 mutants.

The second problem, phenotypic variation, is shared by shRNA depletion and mutant alleles (Waddington, 1942).

Another link between a potential plant heterotrimeric G protein and LRR-RLKs was demonstrated by the observation that gpa1 mutants share the same level of insensitivity to GAs during seed germination as mutants for the LRR-RLK protein BRI1 [ 33].

Furthermore, agmo-1 and the BH4-deficient mutants share a common phenotype of sensitivity to bacterial infection by Leucobacter Verde1.

Our previous studies had demonstrated defects in both LTP and LTD in the D36 mutants and here we demonstrate that, despite similar PKA delocalization in KO and D36 mice, the electrophysiological phenotypes associated with the D36 mutant are not shared by the KO.

For two mutants, or990 ts and or1247 ts, a different mis-sense mutation was found in the same gene in both mutants; this was the only candidate gene shared by both mutants, and the two mutants failed to complement each other (Table S1).