Sentence examples for mutants raised the from inspiring English sources

The increase in NMJ branch numbers in both wnt2 and fz3 mutants raised the possibility that the two genes may function in the same signaling pathway to regulate synaptic growth.

The lack of Atg9 accumulation on Ref(2 P aggregates in Atg18 mutants raised the possibility that Atg18 may facilitate Atg9 recruitment to Ref(2 P.

The similar phenotypes of the mutants raised the possibilities that the causative genetic elements for these mutants were: (1) alleles of the same gene with different grades of expression (Ede and Kelly, 1964b); (2) separate genetic insults within the same pathway; or (3) completely separate defects.

Our finding that lumen fusion is also defective in γCOP null mutants raises the question whether the lumen fusion process is indirectly affected due to γCOP's general effect on luminal protein secretion, or whether the proper functioning of the exocyst complex and the lumen fusion process are affected more directly by the absence of COPI-mediated vesicle trafficking.

This phenotype is very similar to the dominant wing phenotype of H mutants, raising the possibility of dominant negative effects.

The loss of optokinetic response of kif5aa mutants raises the question on which level of the visual pathway the visual information processing is perturbed.

The initiation of differentiated organs in the place of the stem cells in zll mutants raises the question whether AGO10 might negatively regulate auxin function.

The recurrent nature of the TNK2P761Rfs*72 and DDR1R570Q mutants raises the possibility that these may be pathogenic events that bestow a selective advantage in endometrial tumorigenesis.

The observation that Osk levels are increased in slimb mutants raises the question of whether Slimb is involved in the spatial control of Osk accumulation or whether it simply acts to limit Osk levels.

The high frequency of arteriovenous crossings in Nrp1 cytoΔ /Δ mutants raises the possibility that these mice could serve as an animal model to elucidate the poorly understood aetiology of BRVO.

Unexpectedly, although SCC was severely disrupted in diakinesis nuclei of spo-11 rec-8 mutants, indicating that DSBs are essential for tethering sister chromatids, the sisters were together in most pachytene nuclei of spo-11 rec-8 mutants, raising the question of whether DSBs are essential for establishing COH-3/4-mediated cohesion.