As shown in Figure 2 right panel, the mutant male produced normal age-specific courtship suppression.
Overall, mutant conidia produced normal melanized appressoria on hydrophobic surfaces, and none of them formed appressoria on noninductive surfaces (Figure 11 and data not shown).
Introduction of the GFP-Sma plasmid into sma− cells rescued the null mutant phenotype and produced normal WT size plaques on bacterial lawns, rather than the enormous plaques seen with the sma− mutant (Fig. 1I, sma−+GFP-Sma), indicating that sma gene knockout was responsible for the altered plaque size.
MSCs targeted at mutant COL1A2 alleles produced normal type I procollagen and formed bone, thereby demonstrating their therapeutic potential.
In the same conditions, the dnc-1 ; mel-28 double mutants produced a normal brood size.
RICESLEEPER2 mutants produced a normal amount of seeds, but RICESLEEPER1 mutant plants produced mostly empty panicles, yielding only very few seeds, indicating a lethal embryo defect.
Both mutants produced apparently normal siliques, seeds (number and size), and wild-type germination rates (data not shown), indicating that AtSRP2 and AtSRP3 are not essential for meiosis under standard growth conditions.
Interestingly, although mir-83 (n4638 ); mir-34 (gk437 ) mutants produced the normal number of self-progeny in our experiments, they nevertheless exhibited reduced numbers of cross-progeny when mated to wild-type males.
Contrary to pronounced reproductive defects shared by fer-4 and lre mutants, llg1 plants had no reproductive phenotype and produced normal amounts of seeds, consistent with negligible LLG1 promoter activity in pollen and ovules.
Instead, Lolle and Pruitt found that 1% to 10% of the offspring produced normal flowers, indicating that at least one copy of the mutant gene had reverted to the nonmutated wild-type.
