Sentence examples for mutants presented here from inspiring English sources

Future analysis of these mutants in stable transfection systems, as well as continued experiments with the phospho-mimicking mutants presented here, will further reveal the mechanism of action that governs transporter C-terminal phosphorylation.

These features, together with utility as highly specific Gal4 drivers, make gene trap mutants presented here especially useful to the research community.

The three SeV HN mutants presented here showed a temperature-dependent decrease in NA activity to undetectable levels when the proteins were expressed at elevated temperature.

However, we tested a variety of kinetochore mutants in addition to the mutants presented here and found no strict correlation between growth rescue by inhibition of PKA signaling and growth rescue on nonglucose carbon sources.

Data on starchless mutants presented here highlight that two factors are critical when comparing oil contents: i) the strain used as a control (i.e. direct progenitor or other strain) and ii) the basis on which data are expressed (i.e. per cell or per dry weight).

Based on the ability of the non-MFS TB domain mutants presented here to secrete and be incorporated into microfibrils, we propose that the phenotypes of SSS and the acromelic dysplasias are not caused by a primary defect in microfibril assembly.

However, the phenotypic characterization of a complete loss-of-function mutant presented here tends to indicate that Toll-9 is not implicated in the antibacterial arm of the immune response.

The myd88 mutant presented here is a valuable model for determining the role of TLR and IL1R signaling and innate immunity in numerous important processes.

Testing CBI A. thaliana mutants as presented here or in other plant cell systems such as habituated poplar cell suspensions with resistance to CBIs [ 6] can aid in identifying common mechanisms across toxins [ 19, 39] and similarities in resistance mechanisms of the mutants.

The steady-state kinetic characterization of the recombinant wt and mutant enzymes presented here revealed significantly reduced (<5%) relative activity at standard conditions for R116W, R167W and R173W (Table 2), as expected [ 15, 21, 41].

Additional genetic studies, such as the chromosomal deletion of the complete stefin gene cluster mapped on mouse chromosome 16 or the cystatin gene cluster mapped on chromosome 2, would be required to potentially uncover and dissect compensation mechanisms that might be occurring in the mutant mouse model presented here.