Sentence examples for mutants points to from inspiring English sources

The presence of endocytic defects in the majority of zebrafish mechanosensory mutants points to a important role of apical endocytosis in hair cell function.

The morphology of these mutants points to a crosslink of floral organ morphology and flower opening [ 26, 33].

The severe disorganization of both tissues in ush mutants points to important cellular functions mediated by Ush.

A drastically reduced expression of CvPI in flowers of diplocalyx bud bloomer mutants points to a central role of this transcription factor in the formation of this flower type.

The culture supernatants that we used for the experiment did have extracellular GP63 but lacked ER chaperone BiP, as shown in Additional file 9. Overall, alteration in extracellular SAP activity in cells expressing META1 mutants points to a role for META1 in Leishmania secretory processes and highlights the importance of the putative hydrophobic pocket to META1's function.

The fact that fspA KO cells are unable to grow on, and also unable to respond to, capsule-deficient bacteria (as the Kp laboratory strain and LM21 Δ wza and Δ wzx mutants) points to a dual recognition mechanism of K. pneumoniae by Dictyostelium: in the absence of capsule, Dictyostelium relies solely on folate sensing for detecting bacteria, and this recognition is mediated via FspA.

Studies of these knockout mouse mutants point to opposing models of Notch3 function in establishing vascular structure.

Analysis of several mouse mutants point to critical roles for the PIKfyve/VAC14/FIG4 pathway in the central and peripheral nervous systems.

Mutants in lipid metabolic processes and membrane organization also exacerbated the growth defects of mps3 SUN domain mutants, pointing to a role for Mps3 in nuclear membrane organization.

The similarity of the phenotypes observed in Isl1 and Shox2 mutants point to a genetic link between these two genes in the development or function of the cardiac pacemaker.

Attenuation of cAMP, cGMP, calcium, and PI 3,4,5 P3 second-messenger responses in the pikI− mutant points to a defect high in the signal transduction pathway.