Sentence examples for mutants most likely from inspiring English sources

Our analysis reveals that the underlying conformations induced by these AT1R mutants most likely represent principally different mechanisms of uncoupling the G protein, which for some mutants may be due to their increased ability to recruit β-arrestin2.

However, the observed effect on Na+ kinetics in the two mutants most likely reflects the direct effect of mutation on Pi binding and translocation.

No RT-PCR product was obtained from any of the three mutant loci (Fig.  1b), confirming that the T-DNA insertions block gene expression and that the mutants most likely represent knock-out alleles.

Under normal conditions (i.e., without β-Yariv treatment), no inhibition is observed in the single mutants, most likely due to gene redundancy, a conclusion supported by the observation that galt2galt5 double mutants show inhibition under normal conditions [ 16].

Ideally, each mutant should be characterised to define the most likely effect it had on TetA(B) folding and activity, thus identifying those mutants most likely to improve the probability of obtaining well-diffracting crystals.

The higher levels of ssDNA generated further from the chromosome end in cdc13-1 pin1Δ mutants, in comparison with cdc13-1 exo1Δ mostnts, most likely accounts for their sustained metaphase arrest following telomere uncapping.

Therefore, the increased levels of PigU in the hfq mutant most likely contribute to the repression of prodigiosin production.

The TcNPR3 protein only partially complemented the npr3 mutant, most likely because of the heterologous nature of the interactions between TcNPR3 and the Arabidopsis defense response machinery.

One can speculate why a LS ferric haem EPR signal is stronger in the valine mutant compared with the tyrosine mutant – most likely because tyrosine is larger allowing lower R84 occupancy.

The three encountered large errors were in two predictions for MALR (discussed below) and one prediction for GAL4 in the Δmig1 mutant (most likely arising from the microarray experiment itself, as discussed above).

The reduced SodA enzymatic activity of the mtsABC mutant, most likely due to low intracellular levels of manganese resulting from defective manganese transport, may account for this defect in ROS resistance.