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These results indicate that the interactive proteomic network defined by previously identified diploid-specific DOX sensitive mutants is less predictive than genetic interconnectivity in identifying additional DOX resistance genes.

Noticeably, the increase of V1 INs in Jag1f/f NesCre mutants is less pronounced than the increase in V2a INs found in Dll1f/f NesCre mutants, being also statistically less significant (t-test, p<0.05 versus p<0.005) (Fig. 2 D, E).

Moreover, ectopic expression of K10 in degenerated hair follicles, a hallmark of Rbpj deletion in the dermal papilla [25], was not detected in Rbpj/Tgfb3-Cre mice at P8 (Figure S1, C), suggesting that the phenotype seen in mutants is less likely due to Notch signaling loss in the dermal papilla.

In practice, however, interpreting the phenotype of double FA mutants is less straightforward.

The reduction of leaf size and fresh weight in wnk8 mutants is less pronounced than in edm2 mutants.

The probability of finding 0/32 DD and EE mutants by chance, given that we found 7/10 AA mutants, is less than 1 x 10- 5.

The C110A and C110V.RTP mutants were less soluble, less stable and showed lower DNA-binding affinity.

Results indicate that docs1 mutants were less sensitive to gravistimulation than control plants.

In this environment, lighter mutants are less successful and darker mutants are more successful than the parental average.

Bocking et al. (1999) reported that cultures of highly branched mutants are less viscous than those of the parental strains.

However, the expression of OsAS2 in the gs1 2 mutants was less affected in the basal portions (Fig. 1).