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However, a genomewide analysis of Notch signaling defective mutants has not been reported.
The molecular basis of the Ts phenotype of the dnaE, dnaG and dnaC mutants has not been fully analyzed.
Except for the TE7 mutant, the Fix− phenotype of the other mutants has not been further detailed.
However, a systematic screen for defective mutants has not been performed to identify factors important for biofilm formation in these strains.
Other LIM domain adaptor proteins such as Paxillin and PINCH have also been proposed to strengthen integrin linkage to the actin cytoskeleton [34], [35], but whether the localization of these molecules is affected in ΔC3GMS mutants has not been analyzed.
Although the virulence of agr deletion mutants has not been reported for models of skin infection, inhibition of agr-mediated quorum sensing blocked abscess formation in mouse models of S. aureus skin infection [39], [40].
Similar(35)
However, such mutants have not been isolated, and the creation of intended plants by T-DNA insertion has not been possible because a disruption mutant of OsIRE1 is not available.
Although these MSP-119 mutants have not been found in natural population, their recognition by polyclonal antibodies from humans naturally infected with malaria is very promising for the future use of MSP-119 mutants in the design of a malaria vaccine.
However, rice ASNase defective mutants have not been isolated yet.
However, to our knowledge, any deg rice mutants have not been reported yet, still less the function.
Although yfgL mutants are hypersensitive to ampicillin, smpA mutants have not been shown to be hypersensitive.
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