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The involvement of different phytohormones (ethylene, jasmonate, salicylic acid) in modulating the singlet oxygen-mediated cell death during stress in Arabidopsis mutants has also been demonstrated earlier (Danon et al. 2005, Ochsenbein et al. 2006).
Investigation of Creb/Atf1 subfamily mouse mutants has also revealed that Creb/Atf1 subfamily members may compensate for one another.
Relaxation of (positive) interference in meiotic mutants has also been observed, despite a general tendency toward linkage tightening [ 67– 67].
Interestingly, a pronounced activation of STAT1, STAT3 and STAT5 originating from ER-associated Flt3 mutants has also been reported [ 26, 45].
The variation in the degree of silencing in the silenced mutants has also been observed in other fungi (Fitzgerald et al. 2004; Mouyna et al. 2004).
Electron microscopy of Sec16 mutants has also shown an absence of the distinct 40 80 nm COPII transport vesicles (Kaiser and Schekman 1990).
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Mutants have also been isolated from oseltamivir-treated patients and from an immunocompromised zanamivir-treated child.
Barcoded Dictyostelium mutants have also been used as a high-throughput method to piece apart electrotaxis mechanisms, through conservation of genes that correlate with electrotaxis hyperresponsitivity42 though this method is not yet feasible in mammalian cells.
Although most genic male sterility is caused by loss-of-function alleles of genes essential for anther and pollen development (see Wang et al. 2013 for reviews), two dominant genic male sterile mutants have also been reported in rice.
Studies on the N' and O intermediates using selected mutants have also shown disparate results.
TS mutants have also been utilized in Drosophila melanogaster [20], although far fewer examples exist and most have been identified fortuitously.
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