Sentence examples for mutants formed normal from inspiring English sources

The conidia and hyphal tips of the ΔpdeL and ΔpdeH mutants formed normal melanized appressoria on inductive surfaces, similar to the wild-type strain (Figure 6A, Table 2).

All of the other mutants formed normal numbers of nitrogen-fixing nodules (data not shown).

In M. grisea Nox1 and Nox2 mutants formed normal looking appressoria but failed to penetrate the host, suggesting a role for Nox-derived ROS in penetration peg formation [ 61].

Therefore, it seemed unlikely that these types of mutations would be the underlying mutations here because all of our mutants form normal asci when outcrossed indicating meiosis is unaffected (data not shown).

Conversely, in nodules infected by the S. meliloti bacA mutant which contain symbiotic cells with undifferentiated bacteroids, only a subset of NCR genes is activated while in other mutants, forming normal symbiotic cells with differentiated bacteroids, NCR genes are activated to a similar extend as in the wild type [ 18, 19].

A compelling candidate for this step is NC homodimerization: mammalian septins that cannot G dimerize are unable to NC homodimerize in vivo (Kim et al. 2012), but NC homodimerization mutants form normal G dimers (McMurray et al. 2011a; Kim et al. 2012).

Srpk, pk7, and gak mutants formed morphologically normal gametocytes (data not shown), and ookinete cultures contained macrogametes that had emerged from their host cells expressing the activation marker P28 (i.e., Figure 4A), suggesting that neither gametocytogenesis nor early events in macrogametocyte activation were responsible for the lack of ookinete production.

The mutant strains developed in a timely manner in shaken suspension under starvation conditions as well as on phosphate agar plates and formed normal fruiting bodies (data not shown).

The mutant forms normal fruiting bodies and eight-spored asci; however, ascospore pigmentation is different from the wild type.

We show that rse and shd are autonomously required in iridophores, as mutant melanophores form normal sized stripes when confronted with wild-type iridophores in chimeric animals.

Whole mount microscopy of mammary glands engrafted with Stat5ab-/ or PrlR-/ epithelium confirms that both mutants form a normal ductal system in the adult virgin host.