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As daf-2 lf ts) daf-2 lf tsgf;ts) mutands exhibit fully penetrant daf-28 gf tsamined daf-28 gf tsmutantsloss of daf-28, ins-4 and ins-6(lf) could rexhibitlate the dauer phenotype.
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While syp mutants exhibit clear deficits in FM1-43 fluorelative relatove to wildtype controls (supplementary material Fig. S2F,G), this is not fully restored in the genomic rescue.
Both mutants exhibit ligand-independent signal transduction (Figure 4A).
Ozz−/− mutants exhibit defects in myofibrillogenesis and myofiber differentiation.
Thus, both mcl1-101 and swi7-H4 mutants exhibit the cos phenotype.
CtpSE277R mutants exhibit defective growth.
aho-3 mutants exhibit abnormality in thermotactic plasticity.
The pkc1-21 and pkc1-14 mutants exhibit a mitotic delay.
hns stpA double mutants exhibit even stronger phenotypes.
These mutants exhibit moderate or no loss in activity.
Furthermore, Coatg26 mutants exhibit a specific defect in invasion.
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