Sentence examples for mutants demonstrated increased from inspiring English sources

The B. cenocepacia D3 and D4 mutants demonstrated increased sensitivity to inhibitory compounds, suggesting an involvement of these proteins in the intrinsic drug resistance of B. cenocepacia J2315.

As rdgA (Drosophila DGK) mutants demonstrated increased lifespan and lowered p-S6K levels (Fig. S3A,B, Supporting information), we examined whether C. elegans dgk-5 mutants also present the similar phenotypes.

Parallel analysis of cerebellum sections from P10 PEX13 brain mutants demonstrated increased levels of MnSOD, consistent with the findings from the PEX13-null cultured neurons, and further supporting an involvement of mitochondria-mediated oxidative stress in pathogenesis.

Mutations at HIV Gag T186S and T242N reduced VRC, however, in advanced disease only the T242N mutants demonstrated increasing VRC, and were associated with compensatory mutations (p = 0.013).

Additionally, our observations that melanization defective color mutants demonstrate increased virulence in the insect wax moth, suggest the potential of using melanization defective mutants of native insect fungal pathogens in the biological control of insect populations.

Additionally, auxin signaling was shown to increase disease symptoms by pathogens such as Botrytis cinerea and Pseudomonas syringae [ 13, 15] and several auxin signaling mutants demonstrate increased tolerance to different pathogens [ 16- 20].

However, for many of the genes altering expression in relation to antibiotic exposure we could not prove a direct involvement resistance, with only 3 of the 8 B. cenocepacia antibiotic resistance gene deletion mutants demonstrating increased antimicrobial susceptibility.

In contrast, after citrate induction, the ΔPat mutant demonstrated increased glyoxylate/TCA flux compared with wild-type/ΔCobB.

The K56-2ΔM0925-7 mutant demonstrated increased susceptibility to the disinfectant chlorhexidine and the antibiotic azithromycin, but no alteration in susceptibility to other antibiotics.

In the rostral cerebral cortex, almost all NeuN-positive cells in the mutant demonstrated increased pS6 antigen (Fig.  2F and G).

Furthermore, the D538G mutant demonstrated increased proliferation and migration in MTT and wound healing assays, respectively, compared with the wild-type protein.