Sentence examples for mutants demonstrate a from inspiring English sources
Fgfr2b-/ mutants demonstrate a genetically reproducible severe anorectal malformation, characterized by a shortened perineal body, an absence of pelvic rectum, and no visible distal recto-urinary fistula as shown in figure.
Figure 3 shows fast increase of algal cells of wild type (curve 1) after the lag-phase (1st day), as measured by F O. PC Mut2 (curve 2) and PC Mut4 (curve 3) mutants demonstrate a lower growth rate during investigation period.
que mutants demonstrate a progressive defect in motor behavior that culminates in abnormal CNS function and accordion behavior.
On the other hand, both the exocyst and brassinosteroid mutants demonstrate a reduced mature cortical cell length compared to wild-type and this reduction is partially due to slower elongation in both cases (Additional file 2).
In this context, it is noteworthy that GALT6 is highly expressed in senescing leaf tissue and that only galt6 mutants demonstrate a significant reduction in the β-Gal-Yariv precipitable AGPs in senescent leaves (Additional file 1: Figure S4 and Table 2).
Both exocyst and brassinosteroid mutants demonstrate a reduced mature cortical cell length and a slower rate of cell elongation in the elongation zone, but the rate of cell elongation is much more dramatically reduced in exocyst mutants, and the final cell length-to-width ratio is also distinct.
The analysis of sporozoite invasion of hepatocytes in vitro as well as development within primary human hepatocytes with P. falciparum Δp52 mutants demonstrates a pattern of attenuation essentially identical to P. berghei mutants unable to express P36P.
Finally, sequence analysis of these four suppressor mutants demonstrated a lack of the vtc1-1 mutation.
Dramatically, at e13.5, Foxc1 −/− mutants demonstrated a twofold reduction of ventricular zone proliferation, compared to e13.5 wild-type animals.
In contrast, Gli2−/− mouse mutants demonstrated a decrease in proliferation mainly in, otherwise, highly proliferative cortical layers [ 38].
Neural crest apoptosis partially underlies these ethanol-induced defects in pdgfra mutants, demonstrating a protective role for Pdgfra.
