As shown in Figure 1A, under resting condition, the turnover rate in VLCAD−/− mice was significantly reduced by 20% in mutants as compared to the littermates.
Finally, RasIn1 and Rasin2 both display selectivity for the G12V mutants as compared with wild-type Ras providing a potential route for mutant selective recognition of Ras.
Looking for an explanation to the loss of biofilm forming ability in the mutants as compared to the wild type, unfolding studies with guanidinium hydrochloride were undertaken and subsequent fluorescence were observed.
The epithelium of the lactiferous sinus appeared thinner in mutants as compared to WT nipples.
Figure S4 shows no significant differences in the basal expression levels of these chaperone-related genes in the mutants as compared to wild-type control seedlings.
Increasing N from 0.05 to 30 mM in medium with 0.5C caused progressively shorter root lengths in the osu1 mutants as compared to WT plants (Figure 2C).
BOS1 transcript accumulation was however lower after inoculation with the prtE and pelDE mutants as compared to that observed with the wild type parent 30 hpi.
Whole body analysis of Lmna−/− mice and WT littermate controls (Figure 1A, left panels) shows dramatically lower bone mass in the mutants as compared WT controls.
This might be related to the fact that the appearance of maceration symptoms are delayed after inoculation with these mutants as compared to wild type inoculation.
Mitochondrial flux registered a striking increase in both directions in pol γ-β1/β2 and tam3/tam9 mutants as compared to control animals (Figure 8A, Videos S1 and S2).
The points lie near the 45° line of unity, indicating no significant difference in the modification of VOR gain by motor learning in the P/Q mutants as compared with wild-type (F2, 299 = 0.558, p>0.50, ANOVA).
