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Exact(7)
Some of these mutants are probably involved in auxin-related processes, and a very few, including rh2, Lrt1, and ral1, seems to be specifically implicated in root-specific mechanisms.
The results presented above indicated that appressoria formed by ΔpdeH mutants are probably defective in penetration.
Therefore, such mutants are probably not suitable for application in cellulosic ethanol production.
These results indicate that the apparently decreased activities of the Tyr mutants are probably due to their decreased molecular activities.
The important implication is that the fixed phenotypic abnormalities observed in Hsp90 mutants are probably related to de novo induced mutations by transposon activation.
We sequenced three lines and we did not find any mutation in the coding sequence of chico, indicating that at least three of our mutants are probably not alleles of chico.
Similar(53)
The remaining 4 mutants were probably incorrectly associated with RP or have functionalities not discovered yet.
Our analysis suggests that the intermediate phenotypes of the pla/slr1 double mutants were probably due to a reduction in cell number.
Hence the PRC observed in our double mutants is probably caused by changes in the circadian pacemaker.
The decrease of concerted integration activity that was observed for the mutants is probably due to the defect in strand transfer activity in this assay.
Therefore, the slightly reduced level of pre-rRNA association of Noc4p variants in these mutants is probably not responsible for their failure to support growth.
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