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Compared to wildtype cells, ubp2Δ mutants are partially sensitive to the drug.
The analysis of HRF showed that dcl2, dcl3 as well as dcl2 dcl3 and dcl2 dcl3 dcl4 mutants are partially impaired in a stress-induced increase in response to drought, heat, cold and UVC.
Moreover, these mutants are partially sterile.
Additionally, PCP defects in ft mutants are partially rescued by loss of D (Mao et al., 2006).
However, hsi2-2 and hsi2-4 mutares are partially resistant to growth inhibition associated with exposure to the DNA methylation inhibitor 5-aza-2′-deoxycytidine.
The bus mutants are partially resistant to bacterial rectal infection by certain worm pathogens, apparently due to alterations in cuticle surface features (Gravato-Nobre et al. 2005).
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The results showed that yellow-green phenotype of egy1 mutants was partially restored by glucose treatment (Fig. 7a).
In addition, our results showed that the yellow-green phenotype, chlorophyll content and ion leakage of egy1 mutants was partially restored after exogenously applied glucose for 5 weeks.
Furthermore, the yellow-green phenotype of egy1 mutants was partially restored by 2 % glucose (Fig. 7), suggesting that the symptom of early senescence was relieved after glucose supplementation.
The premature lethality of yata mutants was partially but definitely rescued by the neuronal expression of yata.
The intermediate levels of LDH release by both point mutants were partially inhibited by the addition of cytochalasin D (black bars, Fig. 6C).
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