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Figure 1 Characterization of the asl3 mutants at 3-leaf stage: (A) WT plants (Jiahua 1) (B) asl3 mutant plants; (C) RNAi transgenic line transformed with pTCK303-dsRNAiASL3; (D) RNAi control; (E) The pigment contents in leaves at 3-leaf stage in asl3 mutants are much lower than that in WT plant.
In addition, ΔssrA and ΔsmpB mutants are much more sensitive to hygromycin than wild type strain.
This is a function of the viral gp150, in that gp150-deficient mutants are much less GAG-dependent than wild-type.
In fact, these mutants are much more affected by treatment that compromises replication progression, suggesting that the critical function of Mus81 Mms4, Yen1, and Slx1 Slx4 in mitotic cells is to assist in the repair of perturbed RFs.
The late-gestation brain vascular defects and micro-hemorrhages in pericyte conditional Foxc1 mutants are much milder that the severe cerebral hemorrhage observed in Foxc1-null mutant embryos at the same developmental stage (Kume et al., 1998).
The strongest genetic interaction between Pol II mutants and GTF alleles was observed when Pol II LOF mutants and strong sua7 downstream shifting alleles were combined (lethality), suggesting that Pol II LOF mutants are much more sensitive to initiation defects than GOF mutants.
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However, the signal intensity for OsAS1 in phloem companion cells of the nodal vascular anastomoses in the gs1 2 mutants was much lower than in the wild-type rice.
For example, the total brain size of the double mutants is much smaller [10], [32].
Statistical analysis shows that the frequency of aberrant patterns in dRecQ414 mutants is much higher than in wild type control.
Although swi3-E31 ctf18Δ and swi3-E39 ctf18Δ cells were viable, these double mutants were much more sensitive to CPT compared to either single mutant (Figure 7C).
Consistent with this, the transcription levels of CaMCA1 in cmp1Δ and crz1Δ mutants were much lower than that in the wild-type cells (Fig. 4C).
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