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All core protein mutants are derived from EL43.
Both mop mutants are derived from a pool of T-DNA insertion lines (Sieberer et al., 2003).
In human cancers, the most common p53 mutants are derived from missense somatic mutations at 'hot-spot' residues located at the DBD.
In this perspective, the various diverse phenotypes of A1 mutants are derived from a failure to alter the promoter specificity during pre-early gene expression.
Therefore, the compound egg chambers in the sie mutants are derived from multiple cystocyte clusters and this phenotype reflects a defective packaging of individual germ line cysts into discrete egg chambers by the somatic follicle cells.
This result implies that immobile UNC-49B receptors observed in frm-3 single mutants are derived from the NLG-1-stabilized pool and that immobilization of these receptors does not require FRM-3.
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No escape mutants were generated from either of the bat-associated viral variants but two neutralization mutants were derived from the WSKV isolate.
Human eIF4AIII mutants were derived from pET-HTG-eIF4AIII [29] and subcloned into pET28a (BamHI site).
The Tn5EZ mutants were derived from S. aureus RN4220, and all 6,725 mutants are contained on individual, 96-well microtiter plates.
The mutants were derived after exposure to the class IIa bacteriocin sakacin P [20], and relevant characteristics are summarized in Table 1.
As these mutants were derived from the AB1157 E. coli strain, we used AB1157 as reference in this particular RT-qPCR study.
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